IFNA1 and infection: Furthermore, the IFN response pathway was highly upregulated in HuCCT1 and RBE cells and in HSCs upon stimulation with type-I IFN as well as upon infection with VSV(MΔ51) when cells were cultured in their own medium, while activation of the IFN response pathway was significantly reduced when HuCCT1 and RBE cells were cultured in HSC-conditioned medium.