In this study, the hypothesis was raised that MUA has higher expression of invadopodia-forming proteins, MT1-MMP, cortactin, Tks-4, and Tks-5, when compared to other subtypes of unicystic ameloblastoma, due to the location of neoplastic cells being present in the cystic capsule. This evidence concerns the gene SH3PXD2B and ameloblastoma.