The assessment of laboratory markers of cardiovascular injury revealed significantly higher mean concentrations of troponin I (Figure 1B), N-terminal prohormone of brain natriuretic peptide—NT-proBNP (Figure 1C), and the product of fibrin degradation, D-dimer (Figure 1D), in the PIMS group as compared to COVID-19 or INFLAM patients. The gene discussed is NPPB; the disease is COVID-19.