(2016) showed that Ang‐(1–9) preserved left ventricular systolic function in a murine model of myocardial infarction and exerted a positive inotropic effect in adult mice cardiomyocytes by increasing calcium transient amplitude and contractility but whether the regulation of calcium handling can account for the peptide's anti‐arrhythmogenic effect should be addressed by future research [15]. This evidence concerns the gene ANG and myocardial infarction.