It hindered the activation of the NF-κB/NLRP3 inflammasome signaling pathway by enhancing Akt and GSK3β expression, leading to reduced cardiac inflammation, enhanced mitochondrial function, reduced cardiomyocyte apoptosis, and decreased vulnerability to ventricular arrhythmias, ultimately enhancing cardiac function (184). Here, NLRP3 is linked to Ventricular arrhythmia.