For example, the mutation of FUS protein at position 156 from glycine (G) to glutamic acid (E) (G156E) was observed in patients with amyotrophic lateral sclerosis (ALS).[111] From in vitro experiments, G156E FUS was found to undergo LST much faster than the wild‐type FUS protein. This evidence concerns the gene FUS and amyotrophic lateral sclerosis.