At present, the specific mechanism and path of human microorganisms’ role in tumor occurrence and development are still unclear, and only a few studies have achieved some results through experimental exploration, such as the CagA protein secreted by H. pylori, which can also interact with E-cadherin of gastric epithelial cells to promote rapid cell proliferation and increase the risk of epithelial cancer [9]. The gene discussed is S100A8; the disease is neoplasm.