MUTYH and acute kidney injury: Overexpression of type 2 human MUTYH, but not type 1 human MUTYH, markedly improved the renal function of cisplatin‐induced AKI mice compared to that of mice injected with vector plasmids, as evidenced by the lower Scr and BUN levels (Figure 4c), lower cisplatin‐induced renal inflammation (Figure 4d; and Figure S4a, Supporting Information), and lower tubular injury scores based on PAS staining (Figure 4e).