MUTYH and acute kidney injury: FA‐induced AKI has been described in humans[36] and an animal experimental model recapitulates all major processes in human AKI, such as renal cell death and inflammation.[37] Our results showed that MUTYH deficiency aggravated FA‐induced DNA damage, mitochondrial dysfunction, cell death, and AKI in mice, which was consistent with findings in a cisplatin‐induced animal model.