MiR‐155, whose expression is induced by ethanol, promotes the release of EVs from liver cells [18], targeting multiple genes of autolysosomal degradation, whereas miR‐223 increases in serum and in neutrophils of alcoholics and mouse models of ALD, where it inhibits the interleukin‐6–p47phox–ROS pathway, limiting cell infiltration and protecting against alcohol‐induced liver injury [35]. Here, NCF1 is linked to alcohol dependence.