For example, one groundbreaking study identified CEBPB and STAT3 as key synergistic regulators of mesenchymal transformation.[20] The PMT process also encompasses stimulation of both NF‐KB and TAZ signaling cascades.[9, 21] Furthermore, interactions between tumor‐associated macrophages and GBM cells within the tumor microenvironment can promote PMT.[22] However, despite extensive research, the molecular mechanisms underlying PMT in GSCs remain poorly understood. The gene discussed is CEBPB; the disease is neoplasm.