HOTAIR plays an oncogenic role in cervical cancer by promoting cell proliferation, migration, invasion and autophagy, inhibiting cell apoptosis, stimulating angiogenesis, accelerating cell cycle progression, and inducing epithelial-mesenchymal transition [31] lncRNA HOTAIR is upregulated in cervical cancer tissues and regulates the expression of several genes including Wnt/β-catenin pathway, p21, several miRNA such as miR206, miR-143-3p, miR-331-3p, miR-148a [15, 101, 102]. This evidence concerns the gene HOTAIR and cervical cancer.