GOLM1 and laryngotracheoesophageal cleft: Compared to CHB and LC patients, the serum GP73 levels for HBV‐ACLF patients were significantly elevated, which may be correlated to the upregulation of GP73 by inflammatory cytokines, such as interleukin‐6, after the severe immune inflammation that induces the massive and sub‐massive necrosis of liver tissues in HBV‐ACLF patients [15, 26].