IL17A and laryngotracheoesophageal cleft: Furthermore, animal studies corroborated these findings by showing that the introduction of Veillonella into the lungs of lung cancer model mice induces an ecological imbalance in the LRT, triggers severe airway inflammation, increases lung tumor burden, activates the IL-17 inflammatory phenotype and checkpoint inhibitors, and influences the progression and prognosis of LC (48, 49).