Almost every cell in this high TIM-3/HAVCR2 expression group was able to induce T-ALL development compared to 1 in 28 in the low-expression group.63 In a related study, Panelli et al. reported that CD117 and CD82 characterized a distinct T-ALL subpopulation enriched in MRD. The gene discussed is HAVCR2; the disease is acute lymphoblastic leukemia.