Conversely, knocking down CKS1B in BxPC-3 and CFPAC-1 cells overexpressing FOXM1 (Supplementary Fig. 7J, 7K) demonstrated that CKS1B knockdown reversed the enhanced tumor growth (Supplementary Fig. 7L), migration (Supplementary Fig. 7M, 7N), and CSCs self-renewal (Fig. 7F) associated with FOXM1 overexpression. Here, CKS1B is linked to neoplasm.