Upon activation, while OxPhos and glycolysis metabolites increased in Mir142+/+BCR-ABL T cells relative to their resting controls, they did not increase in activated Mir142−/−BCR-ABL T cells vs resting Mir142−/−BCR-ABL T cells (Supplementary Fig. 11b, c; Supplementary Data 2 and 3), suggesting an impaired metabolic reprogramming during T cells’ activation in the Mir142−/−BCR-ABL BC mouse. This evidence concerns the gene ABL1 and breast cancer.