By inhibiting the release of cytochrome C from mitochondria, it makes the downstream related cascade reaction unable to be activated and inhibits apoptosis, thereby reducing the inflammatory cells in the lungs of asthmatic mice, reducing the inflammatory response and alleviating asthma attacks.[34,35] CASP3 is an important protease of the Caspase family, which can reflect the apoptosis of bronchial epithelial cells. Here, CYCS is linked to asthma.