TNFRSF9 and neoplasm: To date, ILA has been discovered that could downregulate glycolysis, NF‐κB, and HIF signaling pathways via the aryl hydrocarbon receptor,[41] and could also transcriptionally enhance function of tumor‐infiltrating CD8 T cells by changing chromatin accessibility.[29] The current study is the first demonstration representing the regulatory impact of ILA on BA metabolism, which improves the comprehension of the role of A. muciniphila and ILA.