The initiation and advancement of atherosclerosis are significantly influenced by vascular inflammation and endothelial dysfunction.[120, 121, 122, 123, 124] The genome‐wide association study (GWAS) study has shown that BACH1 resides in the proximity of a risk locus (rs2832227) for coronary artery disease (CAD).[125] Our recent research has shown that this risk variant was related to BACH1 gene expression in patients’ carotid plaques. This evidence concerns the gene BACH1 and inflammation.