Restenosis after vascular surgery is mainly caused by intimal hyperplasia, which occurs upon vessel injury and involves inflammation, VSMCs dedifferentiation, migration, proliferation, and matrix secretion into the intima.[128, 129, 130, 131] The genetic variant rs73193808 associated with CAD has allele‐specific enhancer activity in human aortic smooth muscle cells (HASMCs),[132] and this variant is closest to the BACH1 gene. The gene discussed is BACH1; the disease is coronary artery disorder.