Second, we relied heavily on the fact that TUG1 has been reported as a miRNA sponge in cerebrovascular diseases and did not consider the possibility that other regulatory mechanisms could also impact TUG‐HuR interactions.[48] Lastly, our study explored the roles of HuR and TUG1 in angiogenesis at specific time points following OGD or MCAO in the designed models, introducing a potential gap regarding clinical practices. The gene discussed is ASPSCR1; the disease is cerebrovascular disorder.