revealed that, in an AngII‐induced model, Cav1+/+ mice exhibited more severe perivascular renal fibrosis and increased expression of vascular cell adhesion molecule‐1 (VCAM‐1) compared to Cav1−/− mice, which exacerbated vascular inflammation (Forrester et al. 2017). The gene discussed is CAV1; the disease is renal fibrosis.