FCGR2A and infection: To subsequently examine their ability to counteract host FcγR activation in the context of infection, we deleted individual vFcγRs or all three of them combined from a recombinant RhCMV strain 68-1 (68-1 R) in which ORF Rh152/151 had been repaired by reversing the single nucleotide change resulting in a premature termination codon (Fig. 4C).