FCGR3A and psoriasis: KO models on FCGR3A and ABO (alpha 1-3-N-acetylgalactosaminyltransferase and alpha 1-3-galactosyltransferase) showed phenotypes such as increased T-cell number, abnormal neutrophil physiology, and aberrant skin morphology suggesting a susceptible role in the pathogenesis of psoriasis (Supplementary Table S10).