Future research should focus on: 1) identifying other targets of PB that may inhibit BC progression; 2) elucidating the molecular mechanism by which PB blocks the interaction between TRIB3 and AKT1, as well as other proteins involved in BC progression; and 3) investigating potential negative physiological signals associated with PB’s activation of the PI3K/Akt pathway in BC inhibition. This evidence concerns the gene AKT1 and breast cancer.