We subsequently found strong evidence of colocalization between the disease GWAS and the ATP11A-eQTL signals (PP4 = 0.996 with IPF GWAS; 0.995 with COVID-19 GWAS; Figures 4B and 4C; Table S10) but weaker support for colocalization between the ATP11A-eQTL signals from the Lung eQTL Study and GTEx lung (PP4 = 0.344; Table S10). The gene discussed is ATP11A; the disease is idiopathic pulmonary fibrosis.