Our focus on the shared genetic variants between critically ill COVID-19 and IPF prompted us to broaden our search to other relevant tissues, revealing that expression of ATP11A in whole blood and DPP9 in fibroblasts may be relevant for the effect of rs12585036 and rs12610495, respectively, during pathogenesis (Figure 3G). This evidence concerns the gene ATP11A and idiopathic pulmonary fibrosis.