In addition, previous findings suggest that EIF3H can interact with and stabilize OGT, enhancing its activity in a deubiquitinase‐dependent manner.[65] This mechanism may similarly operate in obesity‐driven TNBC, amplifying OGT activity and YAP O‐GlcNAcylation through the hexosamine biosynthetic pathway. This evidence concerns the gene YAP1 and obesity due to melanocortin 4 receptor deficiency.