Treatment of glioblastoma cells with MA led to the inhibition of GSK3 kinase, resulting in the downregulation of SRSF1/5, PTPB1, and hnRNP with decreased levels of anti-apoptotic genes such as BCL2, BCL-xL, Survivin, MCL1, and BMI1 while increased in Anxa7, a tumor suppressor gene (Yadav et al., 2014). This evidence concerns the gene BCL2 and glioblastoma.