A link with certain autoimmune diseases, such as primary biliary cholangitis, autoimmune hepatitis, primary sclerosing cholangitis, and autoimmune thyroid diseases, has been recognized in the increased intestinal permeability, the amplification of inflammation through portal circulation, the molecular mimicry between bacterial antigens and epitopes recognized by antimitochondrial antibodies, the aberrant T cell homing to the liver, and tTG reactivity with thyroid tissue in CeD patients, depending on the specific autoimmune condition considered [72]. The gene discussed is TGM2; the disease is cranioectodermal dysplasia.