Reduction in the activity of DNMTs, HMTs, and HDACs, and activation of HATs in cervical tumor cells [66]Regulation of different miRNAs (like miR-145 and miR-146) and regulatory axes (such as miR-22/WNT1/β-catenin and p53/miR-34a/SIRT1) in several tumor cell lines [7]. This evidence concerns the gene TP53 and neoplasm.