SP1 and cancer: PPARƔ/Sp1 interactions were also involved in decreased fatty acid synthesis [110]; induction of adipocyte triglyceride lipase (Atgl); induction of acetyl-CoA synthetase (AACS); and decreased follistatin, KDR, angiotensin type 1 receptor (AT1R) and the thromboxane receptor in non-cancer cell lines [110,111,112,113,114,115,116].