CD36 and endometriosis: Reduced THBS2 expression in epithelial cells stimulated with endometriosis-related PNX-14 concentrations may be responsible for the disrupted THBS2-CD36 signaling axis and impaired phagocytosis of the ectopic epithelium by macrophages, which contributes to the maintenance and proliferation of the endometrium outside the uterus [44,45].