BCL2 and glioblastoma: These data are consistent with the ability of STLs to modulate the expression of Bcl-2 family proteins that control mitochondrial membrane permeability in glioblastoma cells [87], including increasing the Bax/Bcl-2 ratio [18,51,52,56,58,62,63,69,85], down-regulating Mcl-1 [67,83], and up-regulating Bak [68].