Similarly, Chesné et al. and Anderson et al. observed elevated levels of IL-17 from Th17 cells in premature infants, which further exacerbated inflammation, resulting in both eosinophilic (IL-4 and IL-13) and neutrophilic (IL-17) asthma, leading to a mixed type of asthma [182,183]. The gene discussed is IL17A; the disease is asthma.