Oxidative stress not only promotes TAMs to secrete a variety of growth factors, such as VEGF and transforming growth factor-β (TGF-β), to promote the proliferation and angiogenesis of ovarian cancer cells but also promotes the secretion of chemokines by TAMs to promote the migration and invasive ability of ovarian cancer cells, which increases their metastatic risk [88,89]. This evidence concerns the gene TGFB1 and ovarian cancer.