Furthermore, we found that the percentage of Ki67+ ILC1s in the liver was significantly lower in the Ncr1gfp/gfp mice compared to the Ncr1+/+ mice after the animals were challenged with AML cells, suggesting that NKp46 plays a role in ILC1 proliferation in response to AML (Fig. 5b). Here, MKI67 is linked to acute myeloid leukemia.