The term autoimmune nodoparanodopathy was first applied to AMAN–AMSAN but later extended to subacute/chronic forms of polyradiculoneuropathy associated with IgG4 antibodies against nodal (neurofascin [NF]-140/186) or paranodal (NF-155, contactin-1/Caspr1) axo-glial proteins. Here, CNTNAP1 is linked to polyradiculoneuropathy.