It has the ability to restrict several pathways involved in the pathological processes of inflammation, such as dampening the secretion of Tumor Necrosis Factor (TNF), Interleukin‐6 (IL‐6), and other cytokines, as well as downregulating the quantity and function of inflammasomes like NLRP3, which is recognized as a potential pathogenic mechanism of COVID‐19 [23, 24, 25]. The gene discussed is TNF; the disease is COVID-19.