It has the ability to restrict several pathways involved in the pathological processes of inflammation, such as dampening the secretion of Tumor Necrosis Factor (TNF), Interleukin‐6 (IL‐6), and other cytokines, as well as downregulating the quantity and function of inflammasomes like NLRP3, which is recognized as a potential pathogenic mechanism of COVID‐19 [23, 24, 25]. Here, IL6 is linked to COVID-19.