The abundance of NET‐forming neutrophils, characterized by MPO+CitH3+ events, indicated that NETs‐induced thromboinflammation played a significant role in AMI pathogenesis and was closely associated with elevated ACSL1 expression.[9] This comprehensive analysis suggests that ACSL1 is not only a pivotal gene linking hyperlipidemia and AMI but also a key player in the thromboinflammatory processes that characterize these conditions. This evidence concerns the gene ACSL1 and hyperlipidemia.