POLR3E and infection: The first contributes to the virulence of pathogen through the degradation of plant RNA [50], while RPC5 codifies for a component DNA-directed RNA polymerase III affecting an innate immune response in plants by synthesizing small RNAs acting as a fungal pathogen effector that silences host target genes to promote infection, a virulence mechanism called cross-kingdom RNA interference (RNAi) [51,67].