Considering inflammation, RABV and DUVV infections expectedly determined the lowest and the highest ability to promote exogenous RNA sensing response (112) (e.g., TLRs genes, Myd88, Mx1, and Mda5), IFN response (e.g., Stat1-2-4, Oas2-3, and Oasl1) and pro-inflammatory cytokines production (e.g., Il6, Il12b, and Il12rb1-2), respectively (113). Here, OAS2 is linked to infection.