Although BPA exposure can activate microglia and astrocytes, triggering the release of inflammatory factors and posing a potential risk for AD’s onset [214], it could be proposed that BPA and its analogs may exert a role on RACK1 expression in astrocytes through GRα and influence the expression of a set of mRNAs encoding inward-rectifying Kir4.1 potassium (K+) channels. Here, RACK1 is linked to Alzheimer disease.