Enrichment analysis of the dataset revealed that, compared with the healthy group, data from patients with gastric cancer were primarily associated with genes involved in reactive oxygen species production and the cell cycle (Figures 4A–C), which revealed significant differences in gene expression between the high and low ITGAX expression groups, particularly in key biological processes such as EMT, inflammatory response, and NF-κB signaling, potentially contributing to tumor progression and immune modulation. This evidence concerns the gene NFKB1 and gastric cancer.