Moreover, the GLUT1 inhibitor BAY‐876 was used to induce disulfidptosis via pharmacological blockade of glucose uptake as the reference.[1a] Similar to the glucose starvation‐induced disulfidptosis model, we observed that GH pre‐treatment enhanced the sensitivity of HCC cells to BAY‐876‐induced disulfidptosis (Figure S2B, Supporting Information). The gene discussed is SLC2A1; the disease is hepatocellular carcinoma.