For example, one subset of DLBCL is termed OXPHOS‐DLBCL; it exhibits enhanced mitochondrial respiration,[20] enhanced fatty acid oxidation, and enhanced cell proliferation fueled by exogenous fatty acids.[21] Another recent study on CD37‐high DLBCL has similarly highlighted the importance of mitochondria and FAO.[22] Accordingly, our results show how inhibiting FAO and lipolysis in BCL resulted in growth suppression (Figure S3A, Supporting Information); this suggests that downregulating lipid metabolism pathways could benefit BCL treatment. Here, CD37 is linked to diffuse large B-cell lymphoma.