Further confirmation was obtained in an isoproterenol-induced myocardial infarction mouse model, where these two LTB4DH inducers—namely calycosin and GA—significantly reduced the levels of myeloperoxidase (MPO) and MDA in cardiac tissues, thereby attenuating the cardiac morphological changes induced by isoproterenol (Cheng et al., 2015). Here, MPO is linked to myocardial infarction.