CPT2 and myocardial infarction: Notably, the carnitine palmitoyltransferase 2 (CPT2), a key protein that facilitates myocardial fatty acid β‐oxidation, showed the most pronounced decrease.[35] However, treatment with HM4oRL significantly enhanced CPT2 expression in the injured myocardium (Figure 5G,H) and reduced serum free fatty acid (FFA) levels following MI (Figure 5F).