SLC38A1 and pulmonary fibrosis: In bleomycin (BLM)-induced pulmonary fibrosis rat lung tissues and transforming growth factor-β1-treated HFL1 cell models, downregulation of miR-150-5p targets SLC38A1 expression, leading to increased expression of inflammatory cytokines, fibroblast activation, and elevated ROS levels (Yang et al., 2020).