[28] demonstrated that MAF1 expression was decreased in a stroke model, and that miR-122 improves the outcome of acute ischemic stroke by targeting MAF1. A possible explanation for the varied trends in MAF1 expression is that MAF1 may have increased compensation in different cells, and the increase in compensation may be related to the damage time; however, this theory requires further study. Here, MAF1 is linked to stroke disorder.