Djokic et al. and Bras et al. both examined CD123 expression in subtypes of B‐ALL and found that CD123 was highest in patients with hyperdiploid karyotypes, while also increased in patients harboring translocations including ETV6::RUNX1, BCR::ABL1, PBX1::TCF3, and MLL1 (KMT2A) rearrangements (MLLr‐ALL).5, 6. Here, BCR is linked to acute lymphoblastic leukemia.