In OC, YTHDC1 increases the stability of phosphoinositide-3-kinase regulatory subunit 1 in an m6A-dependent manner, and subsequently inhibits glucosidase II alpha subunit expression in N-glycan biosynthesis through signal transducer and activator of transcription 3 signaling, thereby suppressing the tumor growth [39]. This evidence concerns the gene YTHDC1 and neoplasm.